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In the classic db/db mouse model of metabolic fatty liver, oral administration of betaine can inhibit the interaction between forkhead box protein O6 and peroxisome proliferator-activated receptor gamma (PPAR), inhibit the expression of primary lipogenic genes, such as fatty acid synthase (FAS) and acetyl-CoA carboxylase, and reduce hepatic lipid accumulation (77, 78)

Control of dendritic cell trafficking in lymphatics by chemokines

Perelman, S

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MMP inhibition: GHK-Cu suppresses MMP-1 (interstitial collagenase) and MMP-9 (gelatinase B) expression in cultured fibroblasts, reducing collagen degradation alongside synthesis promotion

NAC treatment increased extracellular total antioxidant power and total thiol molecules and enhanced intracellular GSH and patients outcome (106)