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Models of invariant natural killer T cells derived from patient PBMCs also could show that the accumulation of Gb3 negatively affected their numbers and cell function [48], impacting even further on the immune system

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doi: 10.4161/cc.6.22.4936

Meanwhile, myeloid-cell specific GPX4 haploinsufficient mice presented with SLE-like manifestations such as spontaneous skin lesions and systemic damage, further validating that ferroptosis of neutrophils serves a critical role in the pathogenies of SLE

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