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Thus, it can be hypothesized that the ability of DSIP to maintain efficiency of oxidative phosphorylation, even in the setting of hypoxia, could make the peptide a subject of interest for conditions such as stroke and heart attack in future research

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When administered in this manner, GHK-Cu appears to be non-toxic and non-irritating with no adverse effects [2]

AHK-Cu reduced PARP cleavage fragments by 77.5%, confirming reduced cell death