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Qiao and colleagues demonstrated that elevated serum TMAO significantly upregulated mRNA expression of pro-inflammatory M1 microglial markers (CD16, CD32, and iNOS) in the TMAO + MPTP experimental model, suggesting a potential mechanism by which high TMAO levels intensify neuroinflammatory processes in PD through M1 microglial polarization (Qiao et al., 2023)

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