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However, when FPN is missing or ferritin function is abnormal, it will cause iron metabolism imbalance, which will lead to intracellular iron overload, lead to excessive Fe 2+ entering the unstable iron pool (LIP) in the cytosol, generate a large number of ROS through Fenton reaction, destroy lipid peroxidation, and eventually lead to ferroptosis ( Therefore, iron chelating agents and nitrogen oxides can inhibit Fenton reactions, such as deferoxamine (DFO) and TEMPO, thus interrupting ferroptosis ( In addition, ferritin autophagy is a process regulated by autophagy-related (ATG) proteins, which is related to the interaction between autophagy and lysosomes, and nuclear receptor coactivator 4(NCOA4) is its special molecule

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is a Cambrian Biopharma employee and owns Cambrian Biopharma stock options

Unlike its fellow JAK inhibitors, deuruxolitinib does not require liver enzyme monitoring

This dose may be considered rather high as compared with other drugs

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