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Disturbances of mitochondrial biology, primarily mitochondrial bioenergetics, have a long history as a correlate of, and contributing factor to, the pathobiology of autism (48, 49)

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High concentrations of ROS can oxidize mitochondrial proteins, lipids, and nucleic acids, cause cell dysfunction, damage the ability of cells to maintain energy levels, cause energy exhaustion, damage and death of neurons, and change the excitability of neurons, thus reducing the threshold of seizures (6, 7)

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272,325 The opposite effects of SIRT1 on cell proliferation have been observed among different cell types or the regulation of different downstream molecules