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Description

Ototoxicity may arise from multiple factors, leading to damage in cochlear hair cells or in cells responsible for homeostatic functions that regulate cochlear hair cell activity

Beyond direct Nrf2 activators, other agents operate through complementary pathways: Glutamine alleviates injury by inhibiting the xanthine oxidase/uric acid axis[140], while compounds like agmatine and naringin exert their effects by enhancing cellular glutathione reserves and reducing lipid peroxidation[141, 142]