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The rate-limiting step in extracellular GSH catabolism is controlled by -glutamyltransferases (GGTs), which cleave the -glutamyl bond in GSH to release glutamate and dipeptide cysteinylglycine

Lutein attenuated methylglyoxal-induced oxidative damage and apoptosis in PC12 cells via the PI3K/Akt signaling pathway

That is, a compound of Formula (I) and any of the agents described above can be formulated together in the same dosage form and administered simultaneously

This review synthesizes cross-disciplinary evidence to propose a novel hypothetical framework: the gut microbiota-ferroptosis axis as a driver of OA progression

International Journal of Stress Management, 12(2):164-176

However, a comprehensive analytical study is required to monitor any oxidative degradation of compounds over the experimental duration