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demonstrates potential in ameliorating paraquat-induced lung injury and fibrosis by mitigating oxidative stress and ferroptosis through the Nrf2/HO-1 pathway, which further underscores the crucial regulatory role of this pathway in the PF progression [116]

Concurrently, transforming growth factor beta (TGF-) and vascular endothelial growth factor (VEGF) signaling in the cartilage microenvironment accelerates vascular invasion

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This process causes NF-B to translocate to the nucleus, where it promotes the transcription of pro-inflammatory cytokine genes (151)

1 H-NMR-based metabolomics provides simple, efficient and inexpensive technical support for detecting the metabolic fingerprints of ESCC