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Translation of the findings from MASLD/MASH animal models to humans Animal models are crucial for better understanding human MASLD pathogenesis, identifying possible therapeutic targets and disease biomarkers, as well as testing novel drugs

In 6-OHDA unilaterally lesioned rats, UA administration increases GSH levels and SOD activity and decreases the accumulation of malondialdehyde in the striatum [171]

In sensitive LN18 cells, we found that xCT knockdown conferred resistance to combined STF-31 and BSO treatment compared to non-targeting control (Fig

Moreover, the E protein has been shown to participate in activating the host inflammasome 56

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