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doi: 10.1158/1940-6207.CAPR-10-0262 16 Committee opinion no

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In models of neuronal damage and cognitive impairment induced by the chemotherapeutic agent MTX or surgical intervention, MSCs-Exos significantly ameliorated oxidative stress, ferroptosis, and neurological function through activation of the Nrf2-ARE or SIRT1/Nrf2/HO-1 pathways [124, 125]

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Dopamine, oxidative stress and protein-quinone modifications in parkinsons and other neurodegenerative diseases