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Notably, acetylcarnitine can act as an acetyl donor [37], supporting the acetylation process of cells through the shuttle mechanism of acetylcarnitine in the absence of conventional acetyl-CoA synthetase enzymes such as ATP-citrate lyase (ACLY) and acyl-CoA synthetase short chain 2 (ACSS2) [38]

after a period of three months, the levels of the lipofuscin-like material return to normal, indicating the action of a significant disposal mechanism

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No peak at 11.67 min and a small peak (6.2iu) at 11.77 min was observed in three consecutive runs