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The metabolic product of PAHs, particularly BPDE, not only forms covalent adducts with DNA, resulting in genomic instability, but also regulates the expression of oncogenes and tumor suppressor genes through epigenetic mechanisms (Li et al., 2007

If testosterone therapy is started in a patient on a blood thinner, more frequent monitoring of coagulation (e.g., INR) and dose adjustments of the anticoagulant may be necessary

The levels of POD were significantly higher after treatment

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Inflammation-adapted liver stiffness values for improved fibrosis staging in patients with hepatitis C virus and alcoholic liver disease