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This highlights the critical role of dissolution kinetics in NiO NP-induced lung toxicity

In conclusion, we propose a model (Figure 5) in which basic lipophilic compounds accumulate in lysosomes and not only induce cell death due to lysosome membrane permeablization at higher concentrations, but also perturb autophagy process as a result of lysosomal dysfunction at relatively lower concentrations

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Raijmakers MTM, Roelofs HMJ, Steegers EAP et al (2003) Glutathione and glutathione S-transferases A1-1 and P1-1 in seminal plasma may play a role in protecting against oxidative damage to spermatozoa

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