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4-HNE is involved in PD pathology by covalently modifying -syn to promote its aggregation, disrupting the ubiquitin-proteasome system, and interfering with dopamine receptor function, and has been found to alter dopamine transport, contributing to dopamine loss (82, 86, 88)

insights from the ESC-EORP-HFA heart failure long-term registry

Gut microbiota bridges the iron homeostasis and host health

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Since FOXC2 is directly activated by P-p38 phosphorylation, inhibition of this pathway by the p38 inhibitor SB203580 can exert the same effect in restoring AR expression [85] (Fig