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T1-P22 Genomic and Proteomic Analysis of Subspecies of Anopheles culicifacies : Decoding the Mechanism of Refractoriness Pradhan S.R

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Impaired glucose metabolism is a hallmark of AD, which limits the ability of neurons to use glucose as their primary energy source, thereby exacerbating neuronal damage and cognitive decline [3]

Among these, the amino-terminal Neh2 domain facilitates interaction with Keap1 is a substrate adaptor protein for the Cullin3 (Cul3)-dependent E3 ubiquitin ligase complex (57)

We compared the magnitude of the impacts with the minimal clinically important difference threshold for weight loss, indicating that green tea drinking may result in a small reduction in body weight compared with water or placebo

Through regulated turnover of CREBH, ERAD orchestrates the rhythmic expression of genes involved in fatty acid oxidation, triglyceride lipolysis, and gluconeogenesis