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We identified notable metabolites that increased or decreased with age (Fig 1A)

This disruption causes a redox imbalance, leading to membrane lipid peroxidation, reactive oxygen species (ROS) accumulation, and ultimately, iron-dependent oxidative cell death [3]

The N-terminal region of native IGF-1 is directly involved in IGFBP binding: the related analog des(1-3)IGF-I simply deletes the first three residues (Gly-Pro-Glu), and this alone sharply lowers IGFBP affinity while leaving receptor binding largely intact

It is a powerful ally against aging

Postscreening follow-up of the Finnish Prostate Cancer Screening Trial on putative prostate cancer risk factors: vitamin and mineral use, male pattern baldness, pubertal development and non-steroidal anti-inflammatory drug use

As a result, the gut microbiota can recover to the state that existed before the perturbation, a phenomenon known as resilience (3, 4)