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This stress is linked to aging, heart disease, diabetes, brain problems, and even cancer because of how antioxidants work

Through the control of the Fenton reaction, iron chelators such as deferoxamine chelate iron and halt lipid peroxidation

The interaction between aPL and targets like 2GPI triggers the activation of endothelial cells, platelets, monocytes, and neutrophils, as well as the complement and coagulation systems, forming a key molecular mechanism underlying APS pathogenesis (Knight and Erkan, 2024)

Another recurring limitation emerges on antioxidant-based interventions in ASD, constrained by a fundamental methodological gap: most clinical studies report behavioral or functional improvements without directly measuring oxidative-stress biomarkers

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