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Description

N Engl J Med 367:18141820

Mechanistically, STING was shown to directly interact with YY1, inhibiting its nuclear translocation and consequently promoting transcription of lipocalin-2 (LCN2)

J Diab Invest 6(4):369370

alone or co-administered with SERT, it mitigated DEX-induced alteration in acetylcholinesterase activity

Discussion The present review provides a comprehensive overview of the potential therapeutic targets of ferroptosis-associated pathways in stroke, providing novel insights into the application of ferroptosis in the treatment of stroke