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These transporters have been associated with azoles efflux but not with echinocandins whose outcome system has not yet been determined [9], [14]

Available reports have demonstrated that the increase of HDACs activity triggers protein deacetylation, leading to the pathogenesis of a plethora of human diseases including epilepsy [37, 38] and memory deficits [39]

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This distinct metabolic remodelling in the cluster 16 region at days 7 and 28 post-stroke showed different phases of metabolic remodelling (Fig

This change can be triggered by aging, sun exposure, and other environmental factors