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If one ignores the traditional caveats of developmental adaptions in knock-out animals, this suggests that ghrelins ability to increase feeding, particularly of food with high fat content, might be dependent on MC3R, with the possibility that ghrelin-induced, feeding-independent adiposity does not

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In prepubertal female rats, a cerebroventricular infusion of IGF1 stimulated the secretion of GnRH and might advance the onset of puberty [15, 18]

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