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Eur Heart J Cardiovasc Pharmacother 9(8):741757 Uthman L, Li X, Baartscheer A, Schumacher CA, Baumgart P, Hermanides J et al (2021) Empagliflozin reduces oxidative stress through Inhibition of the novel inflammation/NHE/[Na+]c/ROS-pathway in human endothelial cells
Key Points More than 100 genetic loci have been associated with ankylosing spondylitis (AS), but together they explain less than 30% of AS heritability Multiple genes involved in antigen processing and presentation are associated with AS Investigation into the cellular sources and regulation of IL-17 production is crucial to understanding the pathogenesis of AS Cells involved in type 3 immunity produce IL-17 and include innate lymphoid cells and T cells Given the efficacy of the anti-IL-17 monoclonal antibody secukinumab in the treatment of AS, other therapies targeting type 3 immunity could also be effective Abstract Over the past 5 years, advances in high-throughput techniques and studies involving large cohorts of patients have led to considerable advances in the identification of novel genetic associations and immune pathways involved in ankylosing spondylitis (AS)
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preserves NAD+ salvage and SAM availability Regulatory status: not approved by the FDA or any other regulatory authority
