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These effects are typically manageable and often subside with time

Bonn, P

However, the mechanisms of these two types of activation remain unclear

Our recently conducted preclinical studies in the penetrating TBI animal model, demonstrated zero toxic effects for IP-administered NACA at the highest concentration (600 mg/kg), supporting a safer profile for its use as a mitochondrial-targeted neuroprotection compound for TBI (unpublished data)

are supported by MRC project grants APP35041 and UKRI526

When the level of intestinal pro-inflammatory factors increases, it promotes the production of the inflammatory response, causing damage to intestinal mucosa tissue cells, destroying the barrier function of the intestinal epithelium, increasing intestinal permeability, making pathogens and endotoxins enter the blood circulation, causing functional damage to more organs, resulting in a systemic inflammatory response of the body (Costantini et al., 2010)