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1 We chose to use CTCAE data as the primary CIPN endpoint because they were available in all trial participants, whereas PRO data 62 were available only in the subset of patients that participated in the DELCaP substudy 17 and because of our concerns regarding the use of PRO data in CIPN biomarker analyses

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A study conducted on human stomach mucosa cells demonstrated that sulforaphane exhibited protective effects against induced oxidative stress at low concentrations, whereas high concentrations led to increased apoptosis and reduced cell viability [25]