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Mock-infected animals were euthanized on day 14

The severe manifestations of mitochondrial encephalopathies have thus far been explained by the vulnerability of neurons to mitochondrial dysfunction, while reactive gliosis is considered a secondary response to the neuronal pathology

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Spearman correlation explored relationships between pretherapy factors, innate-like CD8+ T cells, and on-treatment HBsAg kinetics

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