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In this study, we found that c-Met and TRK were both activated in gastric cancer cells MKN45, indicating that blocking c-Met and TRK could be an effective strategy for treating gastric cancer

Nestler and the other anonymous reviewer(s) for their contribution to the peer review of this work

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Under inflammation, glia cells become activated, leukocytes are recruited across the BBB, and the BBB itself breaks down, with alterations in paracellular and transcellular transport and tight junction protein expression [63,64,65,66,67]

Scatter factor/hepatocyte growth factor protects against cytotoxic death in human glioblastoma via phosphatidylinositol 3-kinase- and AKT-dependent pathways