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Clinically, many late-onset MADD patients respond dramatically to high-dose riboflavin (100400 mg/day), confirming residual enzyme function amenable to cofactor restorationclassifying MADD as a prototype FAD-responsive metabolic myopathy (70)

Firoozrai, H

Most GHI cases have autosomal recessive inheritance and in the vast majority a molecular defect has been identified involving homozygous or compound heterozygous mutations (Lupu et al., 2001)

2016;44(9):2292-8.DOI: 10.1177/0363546516650885 McAlindon TE, LaValley MP, Harvey WF, et al

It can remove multiple oxidants and is rapidly recycled back to its active form by co-factors in your body like glutathione (GSH)