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Supplemental Information, Table S3), supporting inflammatory activation in manifest disease

Chronic hyperglycemia in DM disrupts normal mitochondrial function leading to excessive ROS generation, advanced glycation end-product (AGE) formation, and activation of pro-inflammatory pathways [29]

The binding between the nickel ion and the agarose support is so strong that it will not be disrupted by high concentration of many commonly used chemicals (see Table 1 for tested chemicals)

Limitations Several limitations of this study should be acknowledged

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