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Under certain nutrient deficient conditions, cancer cells undergo cell death whereas normal healthy cells survive because they are more metabolically flexible ( SLC7A11 mediated cystine uptake is crucial to main redox homeostasis and biomass incorporation in cells ( Figure 2 )

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Moreover, in hemochromatosis patients, homozygous for the C282Y mutation in the HFE-associated gene, silibinin at a dose of 140 mg inhibited the iron absorption by 43 % [91, 92]