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This action protects alveolar epithelial cells against inflammation, injury, and apoptosis via the repression of prolyl hydroxylase 2/HIF-1/MAPK signaling, suggesting its potential in treating COPD and pulmonary fibrosis (Guan et al., 2020)

Prion-like propagation of mutant SOD1 misfolding and motor neuron disease spread along neuroanatomical pathways

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Conclusions and Future Perspectives The pathogenesis of brain aging can be attributed to three interrelated pathological processes: the progressive accumulation of intracellular oxidative stress, dysregulation of energy metabolic networks, and the persistent deterioration of mitochondrial function

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