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An excessive increase in glutamate contributes to glucose-toxicity in -cells via activation of pancreatic NMDA receptors in rodent diabetes

subsequently, through gene function enrichment analysis (such as Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway analysis), clarifying the regulatory roles of target genes in biological processes, molecular functions and signaling pathways

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In the presence of glutathione reductase and NADPH, GSSG is reduced back to GSH, allowing continuous cycling and amplification of the signal

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