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[DOI] [PubMed] [Google Scholar] Gupta D., Lis C.G

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Unlike apoptosis, which is frequently bypassed in glioblastoma through mutations in p53, upregulation of anti-apoptotic proteins (BCL-2, MCL-1), or enhanced DNA repair mechanisms, ferroptosis circumvents these resistance pathways by inducing oxidative membrane damage independent of caspase activation [4, 27]

(560) provided compelling evidence of this relationship, demonstrating a direct link between mucosal adhesion and pathogenic potential in Lactobacillus species

2004;17(Suppl 8):S109

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