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Understanding of mechanisms responsible for cancer cell ferroptosis may help design new drugs for targeted cancer therapy

Research has examined its role in: LHCGR receptor binding, biased agonism, and intracellular cAMP/PKA signalling studies Leydig cell steroidogenesis and testosterone synthesis pathway research HPG axis regulation and gonadotropin interaction studies in male and female models Spermatogenesis, intratesticular testosterone, and male fertility biology research Trophoblast invasion, placentation, and endometrial ECM remodelling investigations Embryo implantation and the embryo-endometrial microenvironment Immune modulation at the maternal-fetal interface regulatory T cells, NK cells, dendritic cells Hyperglycosylated hCG (hCG-H) isoform biology and early pregnancy signalling VEGF-driven angiogenesis and corpus luteum rescue pathway research Comparative LH vs hCG biased agonism and receptor signalling kinetics studies HCG and Corpus Luteum Research Research has shown that rising systemic hCG levels cause a very rapid elevation of serum progesterone, reflecting rescue of the corpus luteum one of hCGs primary endocrine functions

Published research protocols vary

Product summary Ingredients: Water, L-Carnitine L-Tartrate, L-Ascorbic acid, Preservatives (Potassium sorbate, Sodium benzoate), Sweeteners (Acesulfame-K, Sucralose), Flavouring, D-pantothenate calcium, Pyridoxine hydrochloride, Chromium (III)-picolinate, Colour (Indigo carmine)

Selenocystine induces oxidative-mediated DNA damage via impairing homologous recombination repair of DNA double-strand breaks in human hepatoma cells