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Compensatory glutamine metabolism promotes glioblastoma resistance to mTOR inhibitor treatment

Kamaly N, Fredman G, Fojas JJ, Subramanian M, Choi WI, Zepeda K, Vilos C, Yu M, Gadde S, Wu J, Milton J, Carvalho Leitao R, Rosa Fernandes L, Hasan M, Gao H, Nguyen V, Harris J, Tabas I, Farokhzad OC

C.WernerE

The rationale for combination builds upon the documented mechanisms of each component

Ser/gly pathway metabolites are required for RT recovery in NSCLC To validate the observations in the plasma of patients with NSCLC and confirm our hypothesis of augmented ser/gly pathway dependency, we performed 13 C 6 -glucose tracing and metabolomics analysis in a panel of 5 NSCLC cell lines, i.e., NCI-H1299, NCI-H125, NCI-H1975, Calu-6 and NCI-H520 to identify the RT related metabolic changes

Statin DILI cases with definite, highly likely or probable causality scores were identified from Drug Induced Liver Injury Network (DILIN) studies between 2004 and 2022