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Furthermore, there was no lethality and BPC-157 appeared to work in a different manner from existing medications known to cause numerous side effects

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MET amplification/overexpression has been shown to lead to dependence on MET for cell survival [15]

The product of MET proto-oncogene acts as a transmembrane tyrosine kinase receptor for HGF [6]

So the term off-label is off here since it does not have current FDA approval