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Funding This work was supported by the National Natural Science Foundation of China (grant numbers 82173424, 81974476, 81673065, 81830096, 81773341) and the Program of Introducing Talents of Discipline to Universities (111 Project, grant number B20017)

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A combination of keywords related to alpha-synuclein, synaptic plasticity and Parkinsons disease was used: alpha-synuclein, neuroinflammation, neurodegeneration, Lewy bodies, Parkinsons disease, striatal dopaminergic transmission, synucleinopathies, synaptopathy, synaptic plasticity, presynaptic, postsynaptic, toxicity, misfolded, aggregation, oligomers, fibrils, animal models, mechanisms, Lewy body dementia, multiple system atrophy, pure autonomic failure, REM behavior disorder

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107,108 This feedback mechanism acts as a regulatory brake, preventing prolonged or excessive activation of mTORC1 in response to sustained insulin signaling