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[ 197 ] Another study suggests that the inhibition of BACH1 attenuates oxidative stressinduced osteoblast apoptosis and impairment via the amplification of NRF2/ARE signaling, highlighting the BACH1/NRF2/ARE regulatory axis in the development of osteoporosis

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This finding is particularly hopeful, as it aligns with a similar trend observed in a study on the activation of Nrf2 with vitamin E and curcumin through NFB/AKT/mTOR/KEAP1 signalling in rat heart 105