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This results in HIF-1 translocation into the nucleus, where in a complex with HIF-1 it activates transcription via binding to hypoxia responsive elements (HRE) in the promoters of corresponding genes (Kim et al., 2021)

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Moreno MC, Campanelli J, Sande P, Senz DA, Keller Sarmiento MI, et al

With repeated copying, those bindings fray, risking loss of critical data

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