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The UPR also suppresses translation of ER client proteins to decrease the overall protein load in the ER, induces autophagy to assist in protein degradation (Yorimitsu et al., 2006), and activates regulated mRNA degradation to diminish protein influx (Hollien et al., 2009)

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CTR1 is the principal mediator of intestinal copper absorption and intestinal-specific knockout of CTR1 has been shown to result in systemic copper deficiency 12

In-vitro and in-vivo studies