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The pathogenesis of melanoma induced by ultraviolet radiation

ROS formation by NADPH oxidase is triggered by toll-like receptor agonists, which stimulate the innate immune system, resulting in a proinflammatory response manifested by the release of proinflammatory cytokines, such as interleukin-6 (IL-6), tumor necrosis factor-alpha (TNF-), tumor growth factor-beta (TGF-), and platelet-derived growth factor (PDGF) (Jomova et al

Stool (bile), urine and sweat are the main elimination pathways for heavy metals

doi: 10.18632/oncotarget.8387 53 PaloriniRSimonettoTCirulliCChiaradonnaF

Western blot analysis

The M1 phenotype, stimulated by lipopolysaccharide (LPS) and interferon- (IFN-), was used as a positive control, whereas the M2 phenotype, induced by interleukin-4 (IL-4), functioned as a negative control