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that AFB1, as the parent compound, inhibited rat liver mitochondrial electron transport at the antimycin A-sensitive site (10) and postulated subsequently (5) that the enhanced liver cell necrosis and acute toxicity observed in the low-protein animals could be due to the low rate of metabolism and clearance of AFB1 with a consequent effect on mitochondrial respiration

Cell 140 (6), 805820

Chem Res Toxicol 24:549558 Starckx S, Batheja A, Verheyen GR, Jonghe SD, Steemans K, Dijck BV, Singer M, Bogdan N, Snoeys J, Vinken P, Sasaki JC, Gompel JV, Guzzie-Peck P, Lampo A, Lammens L (2013) Evaluation of miR-122 and other biomarkers in distinct acute liver injury in rats

Antidiabetic potential of medicinal plants and their active components

Patients with advanced GI cancers may present with a higher incidence of cachexia given the aggressive nature of chemotherapy, the nutritional deficiency caused by the malignancy, and the relative proximity of localised inflammation and systemic responses

The argument, in a nutshell: Why allow products to be dispensed from compounding pharmacies that, at best, don't work, and at worst, could cause serious health problems