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Treatment of PHGDH-deficient cells with carboplatin or doxorubicin at IC50, respectively, results in elevated mitochondrial ROS and enhanced apoptotic effects

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In summary, while research on phenolic acids in PD is still emerging, compounds like paeoniflorin and gallic acid exemplify how these polyphenols can intervene in PD pathogenesis by modulating neurotransmitter systems, enhancing protein clearance mechanisms, and protecting neurons from oxidative/inflammatory injury