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Herein, we demonstrate that the disruption of the intestinal barrier function occurs before the onset of the inflammatory phase of autoimmune murine and human arthritis identifying it as checkpoint for the transition from autoimmunity to inflammation

Cagrilintide activates the amylin receptor (a CALCR-RAMP complex), contributing gastric-emptying delay, central appetite suppression through area postrema activation, and glucagon inhibition

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