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Current reports suggest that, during the interplay between these two transcription factors, NRF2 appears to be an upstream regulator of HIF

These interactions for 1j might promote tighter binding affinity compared to the endogenous substrate NCA, further indicated by a much lower calculated Vina docking score for 1j (8.1 vs for NCA) that suggests improved energetic interactions for 1j in the NNMT active site

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