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43 Although p105 deficiency has no T-cell intrinsic effect on Th17 cell differentiation, the aberrant activation of NF-B renders innate immune cells hyperresponsive to TLR stimulation for production of IL-6, a major cytokine-promoting Th17 differentiation

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90 As described above, NF-B promotes Th17 differentiation both indirectly through induction of inflammatory cytokines, IL-1, IL-6 and IL-23, in innate immune cells and directly regulates Th17 lineage transcription factors in T cells