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Although the cytotoxic effects of gliotoxin on mammalian cells have been extensively investigated, and yeast have been deployed as a model system to study this interaction [19], no direct investigation of any self-protective mechanism used by A

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The neurogenesis hypothesis of depression may therefore be better described as the neuroplasticity hypothesis of depression a broadening of concept that would include plasticity within the cell, such as mitochondrial activity

2F), indicating that FSP1 + macrophages drive proinflammatory responses in liver fibrosis