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Examining the immunomodulatory impact of pre-existing proinflammatory conditions, such as stress, on subsequent infection, rats subjected to chronic psychosocial stress followed by systemic exposure of polyriboinosinic-polyribocytidilic acid (polyl:C) to mimic infection, experienced prolonged and increased pain sensitivity, or allodynia, and depressive-like behavior, as compared to control animals, as well as enhanced microglia activation (Chijiwa et al., 2015)

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Further research is needed to assess the exact effect of Myo in thyroid pathologies to justify its potential commonness of administration in future

SARS-CoV-2 infection generates massive ROS production and the excessive oxidative damage is responsible for cytokine storm, undermined immunity, tissue damage and severe lung disease causing ARDS and death (Silvagno et al., 2020)

75 used behavioral and electrophysiological techniques to assess the protective effect of N-acetyl cysteine on learning and memory in an A-induced AD model in adult male rats

This breakdown is primarily caused by fungal enzymes, including pectic enzymes that include de-esterifying (pectin methylesterase) and depolymerizing (pectin lyase, polygalacturonase, and pectate lyase) enzymes [12]