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These CAFs-derived molecules, IGF1/2, CXCL12 and beta-hydroxybutyrate, increased ROS level after radiation, thereby enhancing protein phosphatases 2A activity, inhibiting mTOR activation, and increasing autophagy in cancer cells (121)

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These receptors can also assemble inflammasome complexes, such as NLR family pyrin domain containing 3 (NLRP3), in coordination with absent in melanoma 2 (AIM2), resulting in the release of pro-inflammatory cytokinesincluding interleukin-1 (IL-1), IL-18, and TNF-and thereby amplifying neuroinflammation (Elinav et al., 2011