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[171] have identified the NRF2/HO-1 pathway as a crucial regulator of the hepatic microenvironment in ferroptosis, with its activation playing a protective role against oxidative stress in intestinal and hepatic cells

In contrast, green nanomaterials derived from renewable resources such as chitosan, cellulose, and marine polysaccharides provide safer, green alternatives

most resolve within 27 days

aeruginosa can release ALOX15 to induce ferroptosis in bronchial epithelial cells, indicating that pathogen-mediated ALOX15 production mediates host cell death and infection

5 Conclusion Collectively, based on the comprehensive evidence reviewed, this work establishes the bidirectional oral-gut axis as a fundamental mediator of systemic health

These results suggest that genotypic differences in lesions elicit distinct responses to the inflammatory milieu generated by dietary constituents and thus, therapeutic strategies involving dietary changes in women with endometriosis may benefit from incorporating molecular analyses of retrieved lesions