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Characterization of timed changes in hepatic copper concentrations, methionine metabolism, gene expression, and global DNA methylation in the Jackson toxic milk mouse model of Wilson disease

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The increase of our understanding of the role of genetic factors in determining the susceptibility to stroke development may provide an insight into the mechanisms of cerebrovascular complications, and help to identify new therapeutic targets and prophylactic strategies to afford neuroprotection promptly

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GHK-Cu elicits in vitro, dose-dependent transcriptional alterations in pathways relevant to extracellular matrix composition